Oral Trenbolone is incredibly hepatotoxic, and even short cycles can take a toll. For this reason, most people prefer injectable Tren steroids. If you are interested in avoiding injections, though, and you want to use Tren oral, use the smallest possible dose that provides results for the shortest period possible. Be sure to also incorporate some liver protection into your cycle, whether you choose to use milk thistle or a specially-formulated liver care product. Overall, though, the consensus is that both oral and injectable Tren provide the same results at the same doses.
Trenbolone acetate also has the ability to increase red blood cell count. With a larger amount of red blood cells , blood oxygenation is enhanced. This allows for enhanced muscular endurance and therefore promotes a faster rate of recovery. Trenbolone acetate is capable of inhibiting glucocorticoids such as cortisol . The properties of glucocorticoid are the opposite of androgens as muscle tissue depletion and fat gain is promoted.  Administration of trenbolone acetate aims to decrease the production of glucocorticoid hormones. Trenbolone acetate’s contribution to feed efficiency, also known as nutrient efficiency is what makes it an attractive AAS used for agricultural purposes. Food is one of the most anabolic substances that any living organism can consume, and therefore with the administration of trenbolone acetate, every nutrient in the body becomes a lot more valuable.  This facilitates an organism's body that is exposed to the AAS to make better use of the nutrients already consumed.   The non-aromatizing nature of trenbolone acetate makes it a very appealing fat-burning agent. Its capability as a body-sculpting AAS is extreme. [ citation needed ] Many fitness models and bodybuilders use trenbolone acetate, particularly when preparing to compete in an upcoming bodybuilding competition. [ citation needed ]
Suppression of testosterone levels by MGA is responsible for its effectiveness in the treatment of conditions like prostate cancer and benign prostatic hyperplasia.   In one study, 120–160 mg/day MGA suppressed testosterone levels in men by 72%.  However, a recovery or "escape" of testosterone levels, gradually returning to near-normal values, has been observed in most men after 2 to 6 /months of MGA therapy, and this has limited the usefulness of the medication.     The combination of high-dosage MGA (40–160 mg/day) and a low oral dosage of an estrogen such as estradiol (– mg/day), diethylstilbestrol (– mg/day) or ethinylestradiol (50 µg/day) is able to suppress testosterone levels into the castrate range in men, maintain this suppression long-term, and achieve equivalent effectiveness to high-dosage estrogen monotherapy in the treatment of prostate cancer with comparatively greatly reduced toxicity and side effects .          In spite of these results however, this combination has been very rarely used to treat prostate cancer in the United States.